We consider inferring the causal effect of a treatment (intervention) on an outcome of interest in situations where there is potentially an unobserved confounder influencing both the treatment and the outcome. This is achievable by assuming access to two separate sets of control (proxy) measurements associated with treatment and outcomes, which are used to estimate treatment effects through a function termed the causal bridge (CB). We present a new theoretical perspective, associated assumptions for when estimating treatment effects with the CB is feasible, and a bound on the average error of the treatment effect when the CB assumptions are violated. From this new perspective, we then demonstrate how coupling the CB with an autoencoder architecture allows for the sharing of statistical strength between observed quantities (proxies, treatment, and outcomes), thus improving the quality of the CB estimates. Experiments on synthetic and real-world data demonstrate the effectiveness of the proposed approach relative to state-of-the-art methodology for causal inference with proxy measurements.

In settings where ML models are used to inform the allocation of resources, agents affected by the allocation decisions might have an incentive to strategically change their features to secure better outcomes. While prior work has studied strategic responses broadly, disentangling misreporting from genuine adaptation remains a fundamental challenge. In this paper, we propose a causally-motivated approach to identify and quantify how much an agent misreports on average by distinguishing deceptive changes in their features from genuine adaptation. Our key insight is that, unlike genuine adaptation, misreported features do not causally affect downstream variables (i.e., causal descendants). We exploit this asymmetry by comparing the causal effect of misreported features on their causal descendants as derived from manipulated datasets against those from unmanipulated datasets. We formally prove identifiability of the misreporting rate and characterize the variance of our estimator. We empirically validate our theoretical results using a semi-synthetic and real Medicare dataset with misreported data, demonstrating that our approach can be employed to identify misreporting in real-world scenarios.

We consider the problem of estimating a causal effect in a multi-domain setting. The causal effect of interest is confounded by an unobserved confounder and can change between the different domains. We assume that we have access to a proxy of the hidden confounder and that all variables are discrete or categorical. We propose methodology to estimate the causal effect in the target domain, where we assume to observe only the proxy variable. Under these conditions, we prove identifiability (even when treatment and response variables are continuous). We introduce two estimation techniques, prove consistency, and derive confidence intervals. The theoretical results are supported by simulation studies and a real-world example studying the causal effect of website rankings on consumer choices.

The do-calculus is a sound and complete tool for identifying causal effects in acyclic directed mixed graphs (ADMGs) induced by structural causal models (SCMs). However, in many real-world applications, especially in high-dimensional settings, constructing a fully specified ADMG is often infeasible. This limitation has led to growing interest in partially specified causal representations, particularly through cluster-directed mixed graphs (C-DMGs), which group variables into clusters and offer a more abstract yet practical view of causal dependencies. While these representations can include cycles, recent work has shown that the do-calculus remains sound and complete for identifying macro-level causal effects in C-DMGs over ADMGs under the assumption that all clusters sizes are greater than 1.

Current parameter-efficient fine-tuning (PEFT) methods have shown superior performance in continual learning. However, most existing PEFT-based methods focus on mitigating catastrophic forgetting by limiting modifications to the old task model caused by new tasks. This hinders backward knowledge transfer, as when new tasks have a strong positive correlation with old tasks, appropriately training on new tasks can transfer beneficial knowledge to old tasks. Critically, achieving backward knowledge transfer faces two fundamental challenges: (1) some parameters may be ineffective on task performance, which constrains the task solution space and model capacity; (2) since old task data are inaccessible, modeling task correlation via shared data is infeasible. To address these challenges, we propose CaLoRA, a novel causal-aware low-rank adaptation framework that is the first PEFT-based continual learning work with backward knowledge transfer. Specifically, we first propose parameter-level counterfactual attribution (PaCA) that estimates the causal effect of LoRA parameters via counterfactual reasoning, identifying effective parameters from a causal view. Second, we propose cross-task gradient adaptation (CaGA) to quantify task correlation by gradient projection and evaluate task affinity based on gradient similarity. By incorporating causal effect, task correlation, and affinity, CaGA adaptively adjusts task gradients, facilitating backward knowledge transfer without relying on data replay. Extensive experiments across multiple benchmarks and continual learning settings show that CaLoRA outperforms stateof-the-art methods.

Deepfake detection models face two critical challenges: generalization to unseen manipulations and demographic fairness among population groups. However, existing approaches often demonstrate that these two objectives are inherently conflicting, revealing a trade-off between them. In this paper, we, for the first time, uncover and formally define a causal relationship between fairness and generalization. Building on the back-door adjustment, we show that controlling for confounders (data distribution and model capacity) enables improved generalization via fairness interventions. Motivated by this insight, we propose Demographic Attribute-insensitive Intervention Detection (DAID), a plug-and-play framework composed of: i) Demographic-aware data rebalancing, which employs inversepropensity weighting and subgroup-wise feature normalization to neutralize distributional biases; and ii) Demographic-agnostic feature aggregation, which uses a novel alignment loss to suppress sensitive-attribute signals. Across three crossdomain benchmarks, DAID consistently achieves superior performance in both fairness and generalization compared to several state-of-the-art detectors, validating both its theoretical foundation and practical effectiveness.

The do-calculus is a sound and complete tool for identifying causal effects in acyclic directed mixed graphs (ADMGs) induced by structural causal models (SCMs). However, in many real-world applications, especially in high-dimensional settings, constructing a fully specified ADMG is often infeasible. This limitation has led to growing interest in partially specified causal representations, particularly through cluster-directed mixed graphs (C-DMGs), which group variables into clusters and offer a more abstract yet practical view of causal dependencies. While these representations can include cycles, recent work has shown that the do-calculus remains sound and complete for identifying macro-level causal effects in C-DMGs over ADMGs under the assumption that all clusters sizes are greater than 1.

We study the problem of selecting covariates for unbiased estimation of the total causal effect.Existing approaches typically rely on global causal structure learning over all variables, or on strong assumptions such as causal sufficiency - where observed variables share no latent confounders - or the pretreatment assumption, which limits covariates to those unaffected by the treatment or outcome. These requirements are often unrealistic in practice, and global learning becomes computationally prohibitive in high-dimensional settings.To address these challenges, we propose a novel local learning method for covariate selection in nonparametric causal effect estimation that avoids both the pretreatment and causal sufficiency assumptions. We first characterize a local boundary that contains at least one valid adjustment set whenever one exists for identifying the causal effect, and then develop local identification procedures to efficiently search within this boundary.We prove that the proposed method is sound and complete. Experiments on multiple synthetic datasets and two real-world datasets show that our approach achieves accurate causal effect estimation while substantially improving computational efficiency.

Infrastructure deterioration poses significant challenges for asset management, yet existing approaches rely on population-averaged models that overlook equipment-specific heterogeneity. We present a novel framework that combines Bayesian hierarchical hazard modeling with causal discovery to identify operational patterns that drive heterogeneous deterioration rates in pump equipment. Our approach first estimates pump-specific random effects $u_i$ using GPU-accelerated No-U-Turn Sampling (NUTS), achieving 3--5$\times$ speedup over CPU implementations. We then employ DirectLiNGAM to discover causal relationships between 22 engineered time-series features and deterioration rates, stratified by positive ($u_i > 0$, faster deterioration) versus negative ($u_i \leq 0$, slower deterioration) random effects. Analyzing 112 pumps with 92,861 observations over 650 days, we uncover striking heterogeneity: the negative group exhibits causal effects 400$\times$ larger than the positive group, with standard deviation (std) showing a strong positive causal effect ($+1.515$) on deterioration rates in low-risk equipment. We validate linearity assumptions through NonlinearLiNGAM comparison and demonstrate practical scalability through GPU acceleration. Our findings enable targeted maintenance strategies by revealing that different operational regimes require fundamentally distinct management approaches, advancing predictive maintenance from population-averaged to heterogeneity-aware decision making.

Unobserved confounding is a fundamental challenge for estimating causal effects. To address unobserved confounding, recent literature has turned to two different approaches -- proxy variables and the use of multiple treatments. The first approach, commonly referred to as proximal causal inference, requires proxies to be assigned to specific asymmetric roles: treatment-inducing proxies (negative control exposures), variables that act as common causes of the treatment and outcome, and outcome-inducing proxies (negative control outcomes). In practice, however, identifying variables that satisfy these asymmetric roles can be difficult depending on the application domain. The second approach, commonly referred to as the ``Deconfounder," deals with multiple conditionally independent treatments. There has been limited progress towards developing a consistent estimation method for this setting. As the primary contribution of this work, we establish that causal effects are identifiable in both settings when the unobserved confounder is categorical under suitable conditions. Our approach builds on a mixture learning perspective: we show that the underlying confounding structure can be recovered by identifying the corresponding mixture distribution. We propose an estimation procedure based on tensor decomposition, which allows consistent recovery of the latent structure and comes with non-asymptotic guarantees. Simulation studies and real data experiments demonstrate that the proposed method performs well even with limited data.