Most real-world networks are too large to be measured or studied directly and there is substantial interest in estimating global network properties from smaller sub-samples. One of the most important global properties is the number of vertices/nodes in the network. Estimating the number of vertices in a large network is a major challenge in computer science, epidemiology, demography, and intelligence analysis. In this paper we consider a population random graph G = (V;E) from the stochastic block model (SBM) with K communities/blocks. A sample is obtained by randomly choosing a subset W and letting G(W) be the induced subgraph in G of the vertices in W. In addition to G(W), we observe the total degree of each sampled vertex and its block membership. Given this partial information, we propose an efficient PopULation Size Estimation algorithm, called PULSE, that accurately estimates the size of the whole population as well as the size of each community. To support our theoretical analysis, we perform an exhaustive set of experiments to study the effects of sample size, K, and SBM model parameters on the accuracy of the estimates. The experimental results also demonstrate that PULSE significantly outperforms a widely-used method called the network scale-up estimator in a wide variety of scenarios.
While they are very useful to diagnose typical cases, it is difficult for them to diagnose complicated cases. Therefore various approaches, such as deeper knowledge representation, case-based reasoning, are proposed in order to overcome this problem. However, they axe not sufficient to solve this problem completely. One reason that they are not so sutticient is that they are lacking one important track of diagnosis that medical experts do when they meet complicated cases. In this paper, we introduce combination of reasoning, planning and learning methods in order to solve this difficulty.
The process of diagnosis involves learning about the state of a system from various observations of symptoms or findings about the system. Sophisticated Bayesian (and other) algorithms have been developed to revise and maintain beliefs about the system as observations are made. Nonetheless, diagnostic models have tended to ignore some common sense reasoning exploited by human diagnosticians; In particular, one can learn from which observations have not been made, in the spirit of conversational implicature. There are two concepts that we describe to extract information from the observations not made. First, some symptoms, if present, are more likely to be reported before others. Second, most human diagnosticians and expert systems are economical in their data-gathering, searching first where they are more likely to find symptoms present. Thus, there is a desirable bias toward reporting symptoms that are present. We develop a simple model for these concepts that can significantly improve diagnostic inference.
Intelligent agents are often faced with the need to choose actions with uncertain consequences, and to modify those actions according to ongoing sensory processing and changing task demands. The requisite ability to dynamically modify or cancel planned actions is known as inhibitory control in psychology. We formalize inhibitory control as a rational decision-making problem, and apply to it to the classical stop-signal task. Using Bayesian inference and stochastic control tools, we show that the optimal policy systematically depends on various parameters of the problem, such as the relative costs of different action choices, the noise level of sensory inputs, and the dynamics of changing environmental demands. Our normative model accounts for a range of behavioral data in humans and animals in the stop-signal task, suggesting that the brain implements statistically optimal, dynamically adaptive, and reward-sensitive decision-making in the context of inhibitory control problems.
We propose an abductive diagnosis theory that integrates probabilistic, causal and taxonomic knowledge. Probabilistic knowledge allows us to select the most likely explanation; causal knowledge allows us to make reasonable independence assumptions; taxonomic knowledge allows causation to be modeled at different levels of detail, and allows observations be described in different levels of precision. Unlike most other approaches where a causal explanation is a hypothesis that one or more causative events occurred, we define an explanation of a set of observations to be an occurrence of a chain of causation events. These causation events constitute a scenario where all the observations are true. We show that the probabilities of the scenarios can be computed from the conditional probabilities of the causation events. Abductive reasoning is inherently complex even if only modest expressive power is allowed. However, our abduction algorithm is exponential only in the number of observations to be explained, and is polynomial in the size of the knowledge base. This contrasts with many other abduction procedures that are exponential in the size of the knowledge base.